By Bradley S. Fleenor, Adam J. Berrones
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Extra info for Arterial Stiffness: Implications and Interventions
05674 32. Fleenor BS, Marshall KD, Rippe C, Seals DR (2012) Replicative aging induces endothelial to mesenchymal transition in human aortic endothelial cells: potential role of inflammation. J Vasc Res 49(1):59–64. 1159/000329681 33. Hamilton CA, Brosnan MJ, McIntyre M, Graham D, Dominiczak AF (2001) Superoxide excess in hypertension and aging: a common cause of endothelial dysfunction. Hypertension 37(2 pt 2):529–534 34. Tan Y, Tseng PO, Wang D, Zhang H, Hunter K, Hertzberg J, Stenmark KR, Tan W (2014) Stiffening-induced high pulsatility flow activates endothelial inflammation via a TLR2/ NF-kappaB pathway.
Similarly, the SardiNIA showed that B-PP increased for both men and women at the entry age of 55 years, however for men the increase was attenuated until 70 years, which there was little change in B-PP after this age . In contrast to men, the average longitudinal rate of change of B-PP for women continued to increase (monotonically) with every decade from age of 30 to beyond 70 years. In short, both B-PP and C-PP in women have shown to increase with advancing age compared with men [25, 30], indicating central blood pressure is a key target to reduce CVD risk in older women.
However, reports have suggested strength training may increase measures of arterial stiffness. For example, Bertovic et al.  compared apparently 34 3 Implications of Arterial Stiffness healthy young males who had regularly strength trained for 12 months to sedentary controls, and discovered that strength trained athletes had higher aortic characteristic impedance and β stiffness. Additionally, strength trained athletes had greater carotid and brachial pulse pressures compared with the sedentary controls .
Arterial Stiffness: Implications and Interventions by Bradley S. Fleenor, Adam J. Berrones